Effects of electroconvulsive shock treatment on muricide induced by thiamine deficiency
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چکیده
منابع مشابه
Effects of histaminergic drugs on muricide induced by thiamine deficiency.
Male Wistar rats maintained on a thiamine deficient diet showed mouse-killing aggression (muricide). On the 30th day of experimental feeding, the incidence of this muricide was about 70%. Intracerebroventricular histamine suppressed the muricide induced by thiamine deficiency in a dose-dependent manner. Histamine H1-receptor blocking agents such as diphenhydramine, promethazine and chlorphenira...
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The activities of pyruvate and 2-oxoglutarate dehydrogenases were determined by a spectrophotometric method according to Massey, that of transketolase deter mined by Racker's method. The values of enzyme activity as well as the thiamine contents in the preparations were compared with those of the animals whose deficiencies were restored by thiamine administration. The thiamine deprivation resul...
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513 July 2015, Vol. 105, No. 7 Encephalopathy caused by thiamine deficiency To the Editor: Antel et al.[1] have drawn attention to the acute encephalopathy caused by thiamine deficiency in non-alcohol-related settings. I would like to highlight a different clinical presentation that ought to be readily recognised as a possible manifestation of this deficiency, namely rapidly progressive leg wea...
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Cocaine addiction is a widespread problem in the United States, lacking an appropriate treatment. Electroconvulsive shock (ECS) may repair malfunctioning dopamine neurons via induction of synaptic plasticity, providing an alternative treatment for addiction. Sprague Dawley rats were trained to seek cocaine, using a conditioned place preference (CPP) procedure. One group of rats received four c...
متن کاملStudies on the physiological functions of thiamine. I. The effects of thiamine deficiency and thiamine antagonists on the oxidation of alpha-keto acids by rat tissues.
Although thiamine has been known and studied for many years, there is still much we do not know about its physiological functions and mechanism of action in biological systems, particularly in animal tissues. Earlier workers have shown that thiamine diphosphate (cocarboxylase) functions as a coenzyme in the metabolism of pyruvate and a-ketoglutarate (l-9). However, the question regarding the im...
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ژورنال
عنوان ژورنال: Japanese Journal of Pharmacology
سال: 1992
ISSN: 0021-5198
DOI: 10.1016/s0021-5198(19)49444-4